ISSN: 0008-5472

Cancer Research Q1 Unclaimed

American Association for Cancer Research Inc. United States

Unfortunately this journal has not been claimed yet. For this reason, some information may be unavailable.

Cancer Research publishes original studies, reviews, and opinion pieces offering significance and broad impact to a diverse audience. Cancer Research seeks manuscripts that offer pathobiological and translational impact to inform the personal, clinical, and societal problems posed by cancer. It has an SJR impact factor of 3.075.

Cancer Research focuses its scope in these topics and keywords: cancer, tumor, cell, growth, breast, cells, receptor, human, factor, promotes, ...

Type: Journal

Type of Copyright: CC BY-NC-ND

Languages: English

Open Access Policy: Open Choice

Type of publications:

Publication frecuency: -

Categories: Cancer Research (Q1) Oncology (Q1)

Price

4542 €

Inmediate OA

NPD

Embargoed OA

1362 €

Non OA

Metrics

3.075

SJR Impact factor

466

H Index

601

Total Docs (Last Year)

1652

Total Docs (3 years)

29715

Total Refs

16421

Total Cites (3 years)

1621

Citable Docs (3 years)

9.24

Cites/Doc (2 years)

49.44

Ref/Doc

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Aims and Scope

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The Proinflammatory Myeloid Cell Receptor TREM-1 Controls Kupffer Cell Activation and Development of Hepatocellular Carcinoma

Transcriptional profiles and stromal changes reveal bone marrow adaptation to early breast cancer in association with deregulated circulating microRNAs

TAp63-regulated microRNAs suppress cutaneous squamous cell carcinoma through inhibition of a network of cell cycle genes

Oncogenic Kinase-Induced PKM2 Tyrosine 105 Phosphorylation Converts Nononcogenic PKM2 to a Tumor Promoter and Induces Cancer Stem-like Cells

Increased Efficacy of Breast Cancer Chemotherapy in Thrombocytopenic Mice

Inhibition of the Sodium/Potassium ATPase Impairs N-Glycan Expression and Function

BRD4 levels determine the response of human lung cancer cells to BET degraders that potently induce apoptosis through suppression of Mcl-1

Platelet-Derived MHC Class I Confers a Pseudonormal Phenotype to Cancer Cells That Subverts the Antitumor Reactivity of Natural Killer Immune Cells

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