ISSN: 0022-1007

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Journal of Experimental Medicine Q1 Unclaimed

Rockefeller University Press United States

Unfortunately this journal has not been claimed yet. For this reason, some information may be unavailable.

Journal of Experimental Medicine (JEM) is a broad journal that publishes papers providing novel conceptual insight into immunology, cancer biology, vascular biology, microbial pathogenesis, neuroscience, and stem cell biology. We are interested in original findings on all aspects of disease pathogenesis, and we are happy to consider papers reporting novel therapeutic approaches. We strongly encourage submissions of human studies. It has an SJR impact factor of 6,237.

Journal of Experimental Medicine focuses its scope in these topics and keywords: cell, cells, t, human, il, cd, factor, production, dendritic, gene, ...

Type: Journal

Type of Copyright:

Languages: English

Open Access Policy: Open Access

Type of publications:

Publication frecuency: -

Categories: Immunology (Q1) Immunology and Allergy (Q1) Immunology and Allergy (Q1) Medicine (miscellaneous) (Q1)

Price

4517 €

Inmediate OA

1839 €

Embargoed OA

- €

Non OA

Metrics

6,237

SJR Impact factor

478

H Index

169

Total Docs (Last Year)

858

Total Docs (3 years)

11168

Total Refs

10764

Total Cites (3 years)

828

Citable Docs (3 years)

12.31

Cites/Doc (2 years)

66.08

Ref/Doc

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Aims and Scope

Best articles by citations

The reconstitution of cell-mediated immunity in the cutaneous lesions of lepromatous leprosy by recombinant interleukin 2.

Immune response-associated production of neopterin. Release from macrophages primarily under control of interferon-gamma.

Highly restricted expression of the thymus leukemia antigens on intestinal epithelial cells.

Inhibition of T Cell Function During Malaria

Monoclonal antibody with hemagglutination, immobilization, and neutralization activities defines an immunodominant, 47,000 mol wt, surface-exposed immunogen of Treponema pallidum (Nichols).

STUDIES ON THE PATHOGENESIS OF RABIES IN INSECTIVOROUS BATS

BRAIN LESIONS OF THE DOMESTIC RABBIT

IgM anti-Fc gamma R autoantibodies trigger neutrophil degranulation.

Preferential V beta gene usage and lack of junctional sequence conservation among human T cell receptors specific for a tetanus toxin-derived peptide: evidence for a dominant role of a germline-encoded V region in antigen/major histocompatibility com

Development of humanized bispecific antibodies reactive with cytotoxic lymphocytes and tumor cells overexpressing the HER2 protooncogene.

Bone marrow transplantation as a strategy for treatment of non-insulin-dependent diabetes mellitus in KK-Ay mice.

ON THE HbetaMATOZOAN INFECTIONS OF BIRDS

SHOW MORE ARTICLES

Epstein-Barr virus regulates activation and processing of the third component of complement.

Improved HSC reconstitution and protection from inflammatory stress and chemotherapy in mice lacking granzyme B

Oligoclonal T lymphocytes in the cerebrospinal fluid of patients with multiple sclerosis.

Coordinate defects in human histocompatibility leukocyte antigen class II expression and antigen presentation in bare lymphocyte syndrome.

Analysis of T cell stimulation by superantigen plus major histocompatibility complex class II molecules or by CD3 monoclonal antibody: costimulation by purified adhesion ligands VCAM-1, ICAM-1, but not ELAM-1.

Enhanced autoantigen expression in regenerating muscle cells in idiopathic inflammatory myopathy

Interaction of glucocorticoids with macrophages. Identification of glucocorticoid receptors in monocytes and macrophages.

THE ETIOLOGY OF RAT-BITE FEVER

The C3b/C4b receptor is recognized by the Knops, McCoy, Swain-langley, and York blood group antisera.

THE IMMUNIZATION OF FOWLS AGAINST MOSQUITO-BORNE PLASMODIUM GALLINACEUM BY INJECTIONS OF SERUM AND OF INACTIVATED HOMOLOGOUS SPOROZOITES

A physical map of the human regulator of complement activation gene cluster linking the complement genes CR1, CR2, DAF, and C4BP.

ROENTGEN RAY INTOXICATION