Oncogene Q1 Unclaimed

Nature Publishing Group United Kingdom
Unfortunately this journal has not been claimed yet. For this reason, some information may be unavailable.

Oncogene aims to make substantial advances in our knowledge of processes that contribute to cancer by publishing outstanding research. We propagate work that challenges standard conjecture and builds on previous studies, in particular those that lead to establishing new paradigms in the etio-pathogenesis, diagnosis, treatment, or prevention of cancers, and in processes that drive metastatic spread, and provide important insights into cancer biology beyond what has been revealed thus far. It has an SJR impact factor of 3,395.

Oncogene focuses its scope in these topics and keywords: cancer, cell, human, expression, tumor, gene, regulates, growth, cells, protein, ...

Type: Journal

Type of Copyright:

Languages: English

Open Access Policy: Open Choice

Type of publications:

Publication frecuency: -

Price

3490 €

Inmediate OA

NPD

Embargoed OA

- €

Non OA

Metrics

Oncogene

3,395

SJR Impact factor

342

H Index

508

Total Docs (Last Year)

1613

Total Docs (3 years)

27383

Total Refs

14178

Total Cites (3 years)

1601

Citable Docs (3 years)

8,64

Cites/Doc (2 years)

53,90

Ref/Doc

Aims and Scope


cancer, cell, human, expression, tumor, gene, regulates, growth, cells, protein, lung, breast, signaling, apoptosis, induces, pathway, rna, therapeutic, erbb, kinase, receptor, p, Oncology, Cancer, Biology, Genetics



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Identification of maspin and S100P as novel hypomethylation targets in pancreatic cancer using global gene expression profiling

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Expression and functional significance of CDC25B in human pancreatic ductal adenocarcinoma

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JMJD6 regulates histone H2A.X phosphorylation and promotes autophagy in triple-negative breast cancer cells via a novel tyrosine kinase activity

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CD95/Fas signaling in human melanoma cells: conditional expression of CD95L/FasL overcomes the intrinsic apoptosis resistance of malignant melanoma and inhibits growth and progression of human melanoma xenotransplants

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Introductory comments

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Retinal dysplasia in mice lacking p56lck

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