Default: Psychological Medicine

ISSN: 0033-2917

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Psychological Medicine Q1 Unclaimed

Cambridge University Press United Kingdom
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Psychological Medicine is a journal indexed in SJR in Psychiatry and Mental Health and Applied Psychology with an H index of 232. It has a price of 1626.56 €. It has an SJR impact factor of 2,465 and it has a best quartile of Q1. It is published in English. It has an SJR impact factor of 2,465.

Psychological Medicine focuses its scope in these topics and keywords: cognitive, disorder, study, risk, symptoms, stress, genetic, psychosis, patients, outcome, ...

Type: Journal

Type of Copyright:

Languages: English

Open Access Policy: Open Choice

Type of publications:

Publication frecuency: -

Price

1626.56 €

Inmediate OA

NPD

Embargoed OA

0 €

Non OA

Metrics

Psychological Medicine

2,465

SJR Impact factor

232

H Index

542

Total Docs (Last Year)

1042

Total Docs (3 years)

29901

Total Refs

7061

Total Cites (3 years)

957

Citable Docs (3 years)

6.03

Cites/Doc (2 years)

55.17

Ref/Doc

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Aims and Scope


cognitive, disorder, study, risk, symptoms, stress, genetic, psychosis, patients, outcome, anxiety, control, depressive, women, eating, offspring, treatment, posttraumatic, social, impact, disorders,



Best articles by citations

The Treatment of Obsessions. By S. Rachman. Cognitive Behaviour Therapy: Science and Practice Series. Edited by D. Clark, C. Fairburn and S. Hollo. (Pp. 162; £22.95.) Oxford University Press: Oxford. 2003.

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Actions taken to cope with depression at different levels of severity: a community survey

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Schizophrenia in Children and Adolescents. Edited by H. Remschmidt. (Pp. 308; £39.95.) Cambridge University Press: Cambridge. 2001.

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Telepsychiatry and E-mail Health. Edited by R. Wooton, P. Yellowlees and P. McLaren. (Pp. 368; £24.95.) Royal Society of Medicine Press: London. 2003.

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Examining the relationship between pubertal stage, adolescent health behaviours and stress

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Improvement of psychic and somatic symptoms in adult patients with generalized anxiety disorder: examination from a duloxetine, venlafaxine extended-release and placebo-controlled trial

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Neurocognitive function in users of MDMA: the importance of clinically significant patterns of use

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Psychotherapy and Counselling in Practice: A Narrative Framework. By D. Tantam. (Pp. 317; £21.95.) Cambridge University Press: Cambridge. 2003.

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The prediction of thoughts of death or self-harm in a population-based sample of female twins

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Where Inner and Outer Worlds Meet: Psychological Research in the Tradition of George W. Brown. Edited by T. Harris. (Pp. 327; £60.00.) Routledge: London, 2000.

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The effect of a nutritional source of tryptophan on dieting-induced changes in brain 5-HT function

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Psychological consequences of road traffic accidents for children and their mothers

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Frequent attenders in secondary care: a 3-year follow-up study of patients with medically unexplained symptoms

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The detection of intentional contingencies in simple animations in patients with delusions of persecution

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Further neuroendocrine evidence of enhanced vasopressin V3 receptor responses in melancholic depression

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Positive symptoms and white matter microstructure in never-medicated first episode schizophrenia

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Predicting admission rates to secure forensic psychiatry services

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Mood congruent memory bias induced by tryptophan depletion

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Evidence for structural and functional abnormality in the subgenual anterior cingulate cortex in major depressive disorder

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A pragmatic cluster randomized controlled trial of an educational intervention for GPs in the assessment and management of depression

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Depression with late onset is associated with right frontal lobe atrophy

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The genetic epidemiology of body attitudes, the attitudinal component of body image in women

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Negative Symptom and Cognitive Deficit Treatment Response in Schizophrenia. Edited by R. S. E. Keefe and J. P. McEvoy. (Pp. 201.) American Psychiatric Press: Washington, DC. 2001.

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We read with great interest the article by Nielen & Den Boer (33, 917-925), who found that patients with obsessive-compulsive disorder (OCD) displayed cognitive deficits consistent with a dysfunction of the dorsolateral-striatal circuit (DLSC) (i.e.

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