Default: Aging Cell

ISSN: 1474-9718

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Aging Cell Q1 Unclaimed

Wiley-Blackwell Publishing Ltd United Kingdom
Unfortunately this journal has not been claimed yet. For this reason, some information may be unavailable.

Aging Cell is a journal indexed in SJR in Cell Biology and Aging with an H index of 140. It is an CC BY Journal with a Single blind Peer Review review system, and It has a price of 2520 €. The scope of the journal is focused on ageing, longevity, lifespan, apoptosis, gerontology. It has an SJR impact factor of 3,103 and it has a best quartile of Q1. It is published in English. It has an SJR impact factor of 3,103.

Aging Cell focuses its scope in these topics and keywords: human, protein, aging, due, drug, drivers, downregulation, diseaseepigenomic, dehydrobutyrine, dehydroalanine, ...

Type: Journal

Type of Copyright: CC BY

Languages: English

Open Access Policy: Open Access

Type of publications:

Publication frecuency: -

Price

2520 €

Inmediate OA

NPD

Embargoed OA

- €

Non OA

Metrics

Aging Cell

3,103

SJR Impact factor

140

H Index

230

Total Docs (Last Year)

473

Total Docs (3 years)

12277

Total Refs

3878

Total Cites (3 years)

463

Citable Docs (3 years)

7,90

Cites/Doc (2 years)

53,38

Ref/Doc

Aims and Scope


human, protein, aging, due, drug, drivers, downregulation, diseaseepigenomic, dehydrobutyrine, dehydroalanine, dysfunction, elegansextracellular, elevated, enhances, extends, fly, functions, generation, genes, degeneration, decrement, decarboxylase, aginglate, akt, alzheimers, anabolic, antioxidationrelated, aßinduced, bulb, caenorhabditis, cells, cellscombined, common, crosslinking, ctrp, death, ageing, longevity, lifespan, apoptosis, gerontology



Best articles by citations

Age-dependent change in reactive oxygen species and nitric oxide generation by rat alveolar macrophages*

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Rebuttal to Hasty and Vijg: 'Accelerating aging by mouse reverse genetics: a rational approach to understanding longevity'

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Senescence rates in patients with end-stage renal disease: a critical appraisal of the Gompertz model

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Specific suppression of insulin sensitivity ingrowth hormone receptorgene-disrupted (GHR-KO) mice attenuates phenotypic features of slow aging

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Commentary on 'Fast anterograde transport of Herpes Simplex Virus: Role for the amyloid precursor protein of Alzheimer's disease' by Prasanna Satpute-Krishnan et al. Aging Cell Vol. 2, Issue 6, 305-318 (2003)

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Accelerated aging-related transcriptome changes in the female prefrontal cortex

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Loss of pregnancy-associated plasma protein A extends lifespan in mice

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Muscle precursor cells isolated from aged rats exhibit an increased tumor necrosis factor-a response

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Lifespan extension by increased expression of the Drosophila homologue of the IGFBP7 tumour suppressor

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Metal Ions and Neurodegenerative Diseases Paolo Zatta, Editor. World Scientific Publishing Co. Pte. Ltd, Singapore, 2004, pp 400

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Modellingin vivoskeletal muscle ageingin vitrousing three-dimensional bioengineered constructs

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Special Issue: Mitochondria and Aging - Facts and Fancies

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SHOW MORE ARTICLES

Alzheimer-related protein APL-1 modulates lifespan through heterochronic gene regulation in Caenorhabditis elegans

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Sir2 deletion prevents lifespan extension in 32 long-lived mutants

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Introduction

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A genetic interaction between RAP1 and telomerase reveals an unanticipated role for RAP1 in telomere maintenance

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Effects of IGF-1 isoforms on muscle growth and sarcopenia

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Superoxide-lowering therapy with TEMPOL reverses arterial dysfunction with aging in mice

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Advanced oxidation protein products induce pre-osteoblast apoptosis through a nicotinamide adenine dinucleotide phosphate oxidase-dependent, mitogen-activated protein kinases-mediated intrinsic apoptosis pathway

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Dysfunction of the unfolded protein response increases neurodegeneration in aged rat hippocampus following proteasome inhibition

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Introduction

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Born this way: Hippocampal neurogenesis across the lifespan

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Short-term starvation stress at young adult stages enhances meiotic activity of germ cells to maintain spermatogenesis in aged male

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Epigenetic gambling and epigenetic drift as an antagonistic pleiotropic mechanism of aging

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