Default: Clinical Science

ISSN: 0143-5221

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Clinical Science Q1 Unclaimed

Portland Press Ltd United Kingdom
Unfortunately this journal has not been claimed yet. For this reason, some information may be unavailable.

Clinical Science is a journal indexed in SJR in Medicine (miscellaneous) with an H index of 161. It has an SJR impact factor of 1,565 and it has a best quartile of Q1. It is published in English. It has an SJR impact factor of 1,565.

Clinical Science focuses its scope in these topics and keywords: human, receptor, protein, factor, muscle, cardiac, sensitivity, type, constituent, coronary, ...

Type: Journal

Type of Copyright:

Languages: English

Open Access Policy:

Type of publications:

Publication frecuency: -

Price

- €

Inmediate OA

NPD

Embargoed OA

- €

Non OA

Metrics

Clinical Science

1,565

SJR Impact factor

161

H Index

106

Total Docs (Last Year)

490

Total Docs (3 years)

8915

Total Refs

2600

Total Cites (3 years)

486

Citable Docs (3 years)

5.24

Cites/Doc (2 years)

84.1

Ref/Doc

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Aims and Scope


human, receptor, protein, factor, muscle, cardiac, sensitivity, type, constituent, coronary, contractility, deletion, diabetes, diabetesaldosterone, diagnosis, disease, diseaserole, communication, colonic, colitisperoxisome, aging, agonism, amplitude, application, diseasestowards, beating, body, breakdown, bronchial, cardiovascular, chronic, ciliary,



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Effect of prothrombin and its activation fragments on calcium oxalate crystal growth and aggregation in undiluted human urine in vitro: relationship between protein structure and inhibitory activity

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Cardiovascular control and plasma catecholamines during rest and mental stress: effects of posture

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Increased phosphoglycerate kinase in the brains of patients with Down's syndrome but not with Alzheimer's disease

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Use of herpes simplex virus type 1 for transgene expression within the nervous system*

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An immunoluminometric assay for N-terminal pro-brain natriuretic peptide: development of a test for left ventricular dysfunction

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Does the menstrual cycle influence the sensitivity of vagally mediated baroreflexes?

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Glucose flux is normalized by compensatory hyperinsulinaemia in growth hormone-induced insulin resistance in healthy subjects, while skeletal muscle protein synthesis remains unchanged

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Impaired cardiovascular responses to baroreflex stimulation in open-angle and normal-pressure glaucoma

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Systemic inflammatory response syndrome without systemic inflammation in acutely ill patients admitted to hospital in a medical emergency

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